[20] |
Lee TH, Park D, Kim YJ, et al. Lactobacillus salivarius BP121 prevents cisplatin-induced acute kidney injury by inhibition of uremic toxins such as indoxyl sulfate and p-cresol sulfate via alleviating dysbiosis [J]. Int J Mol Med, 2020, 45(4): 1130-1140.
|
[21] |
Fernandes SM, Watanabe M, Vattimo MFF. Inflammation: improving understanding to prevent or ameliorate kidney diseases [J]. J Venom Anim Toxins Incl Trop Dis, 2021, 27:e20200162.
|
[22] |
Karbach SH, Schönfelder T, Brandão I, et al. Gut microbiota promote angiotensin II-induced arterial hypertension and vascular dysfunction [J]. J Am Heart Assoc, 2016, 5(9): e003698.
|
[23] |
Hu J, Luo H, Wang J, et al. Enteric dysbiosis-linked gut barrier disruption triggers early renal injury induced by chronic high salt feeding in mice [J]. Exp Mol Med, 2017, 49(8): e370.
|
[24] |
Al-Harbi NO, Nadeem A, Ahmad SF, et al. Short chain fatty acid, acetate ameliorates sepsis-induced acute kidney injury by inhibition of NADPH oxidase signaling in T cells [J]. Int Immunopharmacol, 2018, 58: 24-31.
|
[25] |
Zheng DW, Pan P, Chen KW, et al. An orally delivered microbial cocktail for the removal of nitrogenous metabolic waste in animal models of kidney failure [J]. Nat Biomed Eng, 2020, 4(9): 853-862.
|
[26] |
Suez J, Zmora N, Segal E, et al. The pros, cons, and many unknowns of probiotics [J]. Nat Med, 2019, 25(5): 716-729.
|
[27] |
Wu H, Singer J, Kwan TK, et al. Gut microbial metabolites induce donor-specific tolerance of kidney allografts through induction of T regulatory cells by short-chain fatty acids [J]. J Am Soc Nephrol, 2020, 31(7): 1445-1461.
|
[28] |
Dong T, Aronsohn A, Gautham Reddy K, et al. Rifaximin decreases the incidence and severity of acute kidney injury and hepatorenal syndrome in cirrhosis [J]. Dig Dis Sci, 2016, 61(12): 3621-3626.
|
[29] |
Christian FK, Hans-Joachim P, Sonja K, et al. Autoimmune renal disease is exacerbated by S1P-receptor-1-dependent intestinal Th17 cell migration to the kidney [J]. Immunity, 2016, 45(5): 1078-1092.
|
[30] |
Nirula A, Nilsen J, Klekotka P, et al. Effect of IL-17 receptor A blockade with brodalumab in inflammatory diseases [J]. Rheumatology, 2016, 55(Suppl 2): ii43-ii55.
|
[1] |
宫铭,陈香美. 急性肾损伤后脑损伤的研究进展[J/CD]. 中华肾病研究电子杂志,2021, 10(1): 40-43.
|
[2] |
Lee SA, Cozzi M, Bush EL, et al. Distant organ dysfunction in acute kidney injury: a review [J]. Am J Kidney Dis, 2018, 72(6): 846-856.
|
[3] |
李江涛,刘怡果,王立瑞,等. 肠道菌群与缺血性急性肾损伤的交互作用[J]. 中华肾脏病杂志,2019, 35(11): 871-875.
|
[4] |
Liu L, Li Q, Yang Y, et al. Biological function of short-chain fatty acids and its regulation on intestinal health of poultry [J]. Front Vet Sci, 2021, 8: 736739.
|
[5] |
秦昊,张健,林俊. 肠道菌群与肾移植的研究进展[J]. 器官移植,2020, 11(4): 516-520, 532.
|
[6] |
Kobayashi T, Iwata Y, Nakade Y, et al. Significance of the gut microbiota in acute kidney injury [J]. Toxins, 2021, 13(6): 369.
|
[7] |
Jang HR, Gandolfo MT, Ko GJ, et al. Early exposure to germs modifies kidney damage and inflammation after experimental ischemia-reperfusion injury [J]. Am J Physiol Renal Physiol, 2009, 297(5): F1457-F1465.
|
[8] |
Al Khodor S, Shatat IF. Gut microbiome and kidney disease: a bidirectional relationship [J]. Pediatr Nephrol, 2017, 32(6): 921-931.
|
[9] |
Gong J, Noel S, Pluznick JL, et al. Gut microbiota-kidney cross-talk in acute kidney injury [J]. Semin Nephrol, 2019, 39(1): 107-116.
|
[10] |
Liu Y, Li YJ, Loh YW, et al. Fiber derived microbial metabolites prevent acute kidney injury through G-protein coupled receptors and HDAC inhibition [J]. Front Cell Dev Biol, 2021, 9: 648-639.
|
[11] |
胡守慈,马红珍. 肠-肾串话机制在急性肾损伤中的研究进展[J]. 临床肾脏病杂志,2021, 21(7): 608-613.
|
[12] |
Andrade-Oliveira V, Amano MT, Correa-Costa M, et al. Gut bacteria products prevent AKI induced by ischemia-reperfusion [J]. J Am Soc Nephrol, 2015, 26(8): 1877-1888.
|
[13] |
Felizardo R, de Almeida DC, Pereira RL, et al. Gut microbial metabolite butyrate protects against proteinuric kidney disease through epigenetic- and GPR109a-mediated mechanisms [J]. FASEB J, 2019, 33(11): 11894-11908.
|
[14] |
Hsu CN, Hou CY, Lee CT, et al. The interplay between maternal and post-weaning high-fat diet and gut microbiota in the developmental programming of hypertension [J]. Nutrients, 2019, 11(9): 1982.
|
[15] |
Yanda MK, Liu Q, Cebotaru V, et al. Histone deacetylase 6 inhibition reduces cysts by decreasing cAMP and Ca2+ in knock-out mouse models of polycystic kidney disease [J]. J Biol Chem, 2017, 292(43): 17897-17908.
|
[16] |
Obeid R, Awwad HM, Rabagny Y, et al. Plasma trimethylamine N-oxide concentration is associated with choline, phospholipids, and methyl metabolism [J]. Am J Clin Nutr, 2016, 103(3): 703-711.
|
[17] |
Nakade Y, Iwata Y, Furuichi K, et al. Gut microbiota-derived D-serine protects against acute kidney injury [J]. JCI Insight, 2018, 3(20): e97957.
|
[18] |
Tang WHW, Wang Z, Kennedy DJ, et al. Gut microbiota-dependent trimethylamine N-oxide (TMAO) pathway contributes to both development of renal insufficiency and mortality risk in chronic kidney disease [J]. Circ Res, 2015, 116(3): 448-455.
|
[19] |
Tomlinson JAP, Wheeler DC. The role of trimethylamine N-oxide as a mediator of cardiovascular complications in chronic kidney disease [J]. Kidney Int, 2017, 92(4): 809-815.
|