[1] |
Ronco C, Bellomo R, Kellum JA, et al. Acute kidney injury [J]. Lancet, 2019, 394(10212): 1949-1964.
|
[2] |
Mercado MG, Smith DK, Guard EL, et al. Acute kidney injury: diagnosis and management[J]. Am Fam Physician, 2019, 100(11): 687-694.
|
[3] |
Peerapornratana S, Manrique-Caballero CL, Gómez H, et al. Acute kidney injury from sepsis: current concepts, epidemiology, pathophysiology, prevention and treatment [J]. Kidney Int, 2019, 96(5): 1083-1099.
|
[4] |
Vázquez-Carballo C, Guerrero-Hue M, García-Caballero C, et al. Toll-like receptors in acute kidney injury [J]. Int J Mol Sci, 2021, 22(2): 816.
|
[5] |
Aluri J, Cooper MA, Schuettpelz LG, et al. Toll-like receptor signaling in the establishment and function of the immune system [J]. Cells, 2021, 10(6): 1374.
|
[6] |
Anthoney N, Foldi I, Hidalgo A, et al. Toll and Toll-like receptor signalling in development [J]. Development, 2018, 145(9): dev156018.
|
[7] |
Mertowski S, Lipa P, Morawska I, et al. Toll-like receptor as a potential biomarker in renal diseases [J]. Int J Mol Sci, 2020, 21(18): 6712.
|
[8] |
Anwar MA, Shah M, Kim J, et al. Recent clinical trends in Toll-like receptor targeting therapeutics [J]. Med Res Rev, 2019, 39(3): 1053-1090.
|
[9] |
Mokhtari Y, Pourbagheri-Sigaroodi A, Zafari P, et al. Toll-like receptors (TLRs): an old family of immune receptors with a new face in cancer pathogenesis [J]. J Cell Mol Med, 2021, 25(2): 639-651.
|
[10] |
Lind NA, Rael VE, Pestal K, et al. Regulation of the nucleic acid-sensing Toll-like receptors [J]. Nat Rev Immunol, 2022, 22(4): 224-235.
|
[11] |
Duan T, Du Y, Xing C, et al. Toll-like receptor signaling and its role in cell-mediated immunity [J]. Front Immunol, 2022, 13: 812774.
|
[12] |
Chen L, Zheng L, Chen P, et al. Myeloid differentiation primary response protein 88 (MyD88): the central hub of TLR/IL-1R signaling [J]. J Med Chem, 2020, 63(22): 13316-13329.
|
[13] |
Bayer AL, Alcaide P. MyD88: at the heart of inflammatory signaling and cardiovascular disease [J]. J Mol Cell Cardiol, 2021, 161: 75-85.
|
[14] |
Owen AM, Fults JB, Patil NK, et al. TLR agonists as mediators of trained immunity: mechanistic insight and immunotherapeutic potential to combat infection [J]. Front Immunol, 2020, 11: 622614.
|
[15] |
Wang J, Wu X, Jiang M, et al. Mechanism by which TRAF6 participates in the immune regulation of autoimmune diseases and cancer [J]. Biomed Res Int, 2020, 2020: 4607197.
|
[16] |
Chen Y, Lin J, Zhao Y, et al. Toll-like receptor 3 (TLR3) regulation mechanisms and roles in antiviral innate immune responses [J]. J Zhejiang Univ Sci B, 2021, 22(8): 609-632.
|
[17] |
Kim SY, Shin S, Kwon M, et al. Suppression of the TRIF-dependent signaling pathway of TLRs by epoxomicin [J]. Arch Pharm (Weinheim), 2021, 354(9): e2100130.
|
[18] |
Peng Y, Zhang X, Wang Y, et al. Overexpression of Toll-like receptor 2 in glomerular endothelial cells and podocytes in septic acute kidney injury mouse model [J]. Ren Fail, 2015, 37(4): 694-698.
|
[19] |
Huang X, Hou X, Chuan L, et al. miR-129-5p alleviates LPS-induced acute kidney injury via targeting HMGB1/TLRs/NF-kappaB pathway [J]. Int Immunopharmacol, 2020, 89(Pt A): 107016.
|
[20] |
Wang QL, Xing W, Yu C, et al. ROCK1 regulates sepsis-induced acute kidney injury via TLR2-mediated endoplasmic reticulum stress/pyroptosis axis [J]. Mol Immunol, 2021, 138: 99-109.
|
[21] |
Jia P, Xu S, Wang X, et al. Chemokine CCL2 from proximal tubular epithelial cells contributes to sepsis-induced acute kidney injury [J]. Am J Physiol Renal Physiol, 2022, 323(2): F107-F119.
|
[22] |
Li H, Qiu D, Yuan Y, et al. Trichinella spiralis cystatin alleviates polymicrobial sepsis through activating regulatory macrophages [J]. Int Immunopharmacol, 2022, 109: 108907.
|
[23] |
Xia S, Lin H, Liu H, et al. Honokiol attenuates sepsis-associated acute kidney injury via the inhibition of oxidative stress and inflammation [J]. Inflammation, 2019, 42(3): 826-834.
|
[24] |
Peng Y, Liu L, Wang Y, et al. Treatment with Toll-like receptor 2 inhibitor ortho-vanillin alleviates lipopolysaccharide-induced acute kidney injury in mice [J]. Exp Ther Med, 2019, 18(6): 4829-4837.
|
[25] |
Jha AK, Gairola S, Kundu S, et al. Toll-like receptor 4: an attractive therapeutic target for acute kidney injury [J]. Life Sci, 2021, 271: 119155.
|
[26] |
Anderberg SB, Luther T, Frithiof R, et al. Physiological aspects of Toll-like receptor 4 activation in sepsis-induced acute kidney injury [J]. Acta Physiol (Oxf), 2017, 219(3): 573-588.
|
[27] |
Wang B, Xu J, Ren Q, et al. Fatty acid-binding protein 4 is a therapeutic target for septic acute kidney injury by regulating inflammatory response and cell apoptosis [J]. Cell Death Dis, 2022, 13(4): 333.
|
[28] |
Gatica S, Villegas V, Vallejos A, et al. TRPM7 mediates kidney injury, endothelial hyperpermeability and mortality during endotoxemia [J]. Lab Invest, 2020, 100(2): 234-249.
|
[29] |
Smith JA, Stallons LJ, Collier JB, et al. Suppression of mitochondrial biogenesis through toll-like receptor 4-dependent mitogen-activated protein kinase kinase/extracellular signal-regulated kinase signaling in endotoxin-induced acute kidney injury [J]. J Pharmacol Exp Ther, 2015, 352(2): 346-357.
|
[30] |
Gao Q, Zheng Y, Wang H, et al. circSTRN3 aggravates sepsis-induced acute kidney injury by regulating miR-578/Toll like receptor 4 axis [J]. Bioengineered, 2022, 13(5): 11388-11401.
|
[31] |
Wu S, Qiu H, Wang Q, et al. Effects and mechanism of lncRNA CRNDE on sepsis-induced acute kidney injury [J]. Anal Cell Pathol (Amst), 2020, 2020: 8576234.
|
[32] |
Zhang Q, Wang L, Wu M, et al. Humanized anti-TLR4 monoclonal antibody ameliorates lipopolysaccharide-related acute kidney injury by inhibiting TLR4/NF-κB signaling [J]. Mol Med Rep, 2021, 24(2): 608.
|
[33] |
Zhao G, Lu S, Li L, et al. Local anesthetic articaine ameliorates LPS-induced acute kidney injury via inhibition of NF-κB activation and the NLRP3 inflammasome pathway [J]. J Biochem Mol Toxicol, 2020, 34(10): e22554.
|
[34] |
Zeng M, Qi M, Wang Y, et al. 5-O-methyldihydroquercetin and cilicicone B isolated from Spina Gleditsiae ameliorate lipopolysaccharide-induced acute kidney injury in mice by inhibiting inflammation and oxidative stress via the TLR4/MyD88/TRIF/NLRP3 signaling pathway [J]. Int Immunopharmacol, 2020, 80: 106194.
|
[35] |
Fan H, Zhao Y, Zhu JH, et al. S-nitrosoglutathione protects lipopolysaccharide-induced acute kidney injury by inhibiting Toll-like receptor 4-nuclear factor-κB signal pathway [J]. J Pharm Pharmacol, 2019, 71(8): 1255-1261.
|
[36] |
Tsuji N, Tsuji T, Ohashi N, et al. Role of mitochondrial DNA in septic AKI via Toll-like receptor 9 [J]. J Am Soc Nephrol, 2016, 27(7): 2009-2020.
|
[37] |
Naito Y, Tsuji T, Nagata S, et al. IL-17A activated by Toll-like receptor 9 contributes to the development of septic acute kidney injury [J]. Am J Physiol Renal Physiol, 2020, 318(1): F238-F247.
|
[38] |
Li H, Sun H, Xu Y, et al. Curcumin plays a protective role against septic acute kidney injury by regulating the TLR9 signaling pathway [J]. Transl Androl Urol, 2021, 10(5): 2103-2112.
|
[39] |
Sun BQ, Sui YD, Huang H, et al. Effect of lncRNA CRNDE on sepsis-related kidney injury through the TLR3/NF-κB pathway [J]. Eur Rev Med Pharmacol Sci, 2019, 23(23): 10489-10497.
|
[40] |
Lin X, Huang H, You Y, et al. Activation of TLR5 induces podocyte apoptosis [J]. Cell Biochem Funct, 2016, 34(2): 63-68.
|
[41] |
Yang X, Yin Y, Yan X, et al. Flagellin attenuates experimental sepsis in a macrophage-dependent manner [J]. Crit Care, 2019, 23(1): 106.
|
[42] |
Wang HF, Li Y, Wang YQ, et al. MicroRNA-494-3p alleviates inflammatory response in sepsis by targeting TLR6 [J]. Eur Rev Med Pharmacol Sci, 2019, 23(7): 2971-2977.
|
[43] |
Jian W, Gu L, Williams B, et al. Toll-like receptor 7 contributes to inflammation, organ injury, and mortality in murine sepsis [J]. Anesthesiology, 2019, 131(1): 105-118.
|